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Postmenopausal hormone therapy and mortality.

Plasma total homocysteine in healthy subjects: Sex-specific relation with biological traits.

Sex-differential genetic effect of phosphodiesterase 4D (PDE4D) on carotid atherosclerosis.

Effect of antihypertensive drug treatment on cardiovascular outcomes in women and men: a meta-analysis of individual patient data from randomized, controlled trials.

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Likewise, women who experienced an early or premature menopause, most commonly due to bilateral oophorectomy, are now known to be at increased risk for cognitive decline or dementia and have been found to derive some protection from taking estrogen replacement.

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Estimates of the risk of cardiovascular death attributable to low-dose oral contraceptives in the United States.

An assessment of risk factors for the complexity of coronary artery disease using the SYNTAX score.

John Lee also states that recent studies have shown that “women who use hormone replacement therapy containing estrogen are more likely to get lupus.

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While parenterally administered medroxyprogesterone acetate inhibits gonadotropin production, which in turn prevents follicular maturation and ovulation, available data indicate that this does not occur when the usually recommended oral dosage is given as single daily doses.

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It can be used for the management of menopausal symptoms such as post menopausal osteoporosis, breast cancer, dysmenorrheal, dysfunctional uterine bleeding and female hypogonadism.

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GnRH antagonist) is an analogue that blocks the GnRH receptor resulting in an immediate drop in gonadotropin (FSH, LH) secretion.

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Laird random-effects model would have been used if there were heterogeneity between the studies.

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